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Clinical observations were recorded during hospitalization. Levels of 45 immune mediators were quantified using multiplex microbead-based immunoassays.Whereas 11.6% of patients had neurological complications, 88.4% displayed mild disease of rash and fever., and IP-10 were observed in ZIKV-infected pregnant women carrying fetuses with fetal growth-associated malformations.Notably, infants with congenital central nervous system deformities had significantly higher levels of interleukin 18 and IP-10 but lower levels of hepatocyte growth factor than those without such abnormalities born to ZIKV-infected mothers.However, the immature ZIKV contains a partially ordered capsid protein shell that is less prominent in other immature flaviviruses.

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14/103 (13.5%) echocardiograms were compatible with congenital heart disease: 5 with an ostium secundum atrial septal defect, 8 had a hemodynamically insignificant small apical muscular ventricular septal defect and one infant with dyspnea had a large membranous ventricular septal defect.Several immune mediators were specifically higher in ZIKV-infected patients, and levels of interleukin 10, interferon gamma-induced protein 10 (IP-10), and hepatocyte growth factor differentiated between patients with or without neurological complications.Interestingly, higher levels of interleukin 22, monocyte chemoattractant protein 1, TNF-?This study identified several key markers for the control of ZIKV pathogenesis.This will allow a better understanding of the molecular mechanisms of ZIKV infection in patients.

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